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A team of researchers ZaidÃn Experimental Station (CSIC) has confirmed that the proteins ‘Vcx1p’ and ‘Vnx1p’ facilitate the transport of sodium and potassium in the yeast cells, which perform this operation in a manner similar to plants . The work may help design strategies for salinity-tolerant crops.
Researchers at the Department of Biochemistry, Molecular Cell Biology and Plant Experimental Station Zaidin (CSIC) have found that two proteins are responsible for transporting potassium and sodium inside the cells of yeast.
In particular, the action of the proteins ‘Vcx1p’ and ‘Vnx1p’ makes it possible to accumulate these mineral nutrients in small sacs called vacuoles, as scientists published in the latest issue of The Journal of Biological Chemistry.
Since yeast and plant cells share many transport mechanisms, the work opens the way for the design of strategies to improve plant nutrition and crop tolerance to salinity, ie the high concentration of sodium in soil and irrigation water.
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Prof. Patrick Provost, Laval University and his team at the Centre Hospitalier Universitaire de Quebec have recently demonstrated the presence of a family of molecules that regulates protein synthesis in blood platelets. Their work was published in the August issue of the journal Nature Structural & Molecular Biology “.
Platelets play an essential role in blood clotting. Their function is performed by proteins. Platelets are devoid of cell nucleus and DNA, the synthesis of these proteins is provided by ribonucleic acids (mRNAs) inherited the cells from which they arise. In general, control of protein synthesis is made from DNA via RNA. But in the absence of DNA, how that control is exercised there? Patrick Provost and his colleagues believe they have found the solution: they are microRNAs, which would ensure that regulation in platelets.
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A recent study conducted by researchers at the Centro de Biologia Molecular “Severo Ochoa” (CSIC-UAM), Universidad Autonoma de Madrid, addresses the question of how the virus of African swine fever takes control of the cell and prevents the same counter viral infection.
It is known that viruses are intracellular parasites able to employ different strategies to control and interfere with cellular mechanisms of defense against infection. One strategy is to hijack the cellular machinery of protein synthesis. Thus, the virus saves the energy of the cell to synthesize its own proteins, and prevents the infected host antiviral synthesize those molecules that could counteract the viral infection.
However, the molecular basis of the mechanisms used by viruses to carry out this type of phenomenon is still not determined.
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